EMF Blocking and even EMF Shielding
Whether you live in an apartment or a house, or you just would like to ensure your home is free of electromagnetic fields, there are a number of ways you can reduce exposure. One of the easiest is to reduce the use of electronic devices. emf blockers can also turn to EMF block paint to stop EMF radiation from entering your house. Another way to shield your home against EMF radiations is to put up a shielding canopy for RF. It is a type made of net that has EMF shielding and is used to block EMFs from entering rooms. emf blocking is to get your home equipped with an enclosure that is conductive. These enclosures are known as Faraday cages.
A number of studies have proven that the non-ionizing RF EMF produces antiproliferative effects on HCC cells. emf blocking that drives AM RF EMF's anticancer activity in vitro is believed to involve down-regulation in cancer-related stem cells. This could explain the long-term response seen in patients suffering from advanced HCC. But, the reason for AM EMF's impact on patients suffering from cancer isn't yet clear.
The effects on the effects of AM RF EMF on HCC tumour growth in vivo was studied in mice. The tumours were divided into 3 groups. The first group was not exposed RF EMF. The second group was exposed RF EMF at the same frequency to the one used by humans. The third group was exposed to RF EMF at HCC-specific modulation frequencies. The effects of HCCMF on the tumours was assessed against the effect of RCF. The results showed that the tumors treated by HCCMF had significant shrinkage. However, tumours treated with RCF didn't show evidence of tumour shrinkage.
The reason for tumour-specific AM RF EMF may be based on the fact that tumor cells require Cav3*2 type voltage calcium channels for their proliferation and down-regulation. AM RF EMF's antiproliferative effects in HCC cells is caused through CACNA1H, a protein that regulates the Ca2+ influx specific to tumors. The results suggest that CACNA1H could have wider implications for the treatment and diagnosis of many cancers.
The tumours in those in the group that were unaffected to EMF from radiofrequency, and fed a standard mouse diet. The tumours in HCCMF HCCMF group were injected with Huh7 cells at the time they were between five and seven weeks old. The tumours were then euthanized after they had a high burden.
The tumours from the three groups also showed distinct growth curves. The tumors treated with HCCMF showed a significant decrease in the size of the tumour after eight weeks. However, tumors that were treated using RCF showed no shrinkage. The difference was substantial. The tumours treated with RCF showed necrosis, which is typical in tumors that have been exposed to RCF. The possibility is that the necrosis is caused by the lack of oxygen in the larger tumors.
In conclusion, the findings indicate an AM-RF EMF exhibits anticancer effects in vitro and in vivo. A number of studies have proven the fact that AM RF EMF produces measurable tumour shrinkage for HCC patients. There is a possibility that AM RF EMF causes these effects because of CACNA1H, a protein that is involved in the process of tissue-specific Ca2+ influx. Furthermore, AM RF EMF may exert a sustained influence on the growth of HCC tumors in the vivo.